Testosterone production, action, and clinical manifestations
In the male body, the hypothalamus secretes gonadotropin-releasing hormone, or GnRH, in a pulsatile fashion to stimulate the release of follicle-stimulating hormone, or FSH, and luteinizing hormone, or LH, from the pituitary gland. LH travels in the bloodstream to the Leydig cells of the testes, where it binds to the LH receptor and initiates a series of biochemical events that convert low-density lipoprotein or LDL cholesterol to testosterone. Testosterone secreted from the testes is carried by the bloodstream to target tissues where it produces its biological effects.
Failure of the testes to make physiological levels of testosterone is called hypogonadism. Hypogonadism due to abnormalities of the testes themselves is called primary hypogonadism, whereas a defect in the hypothalamic-pituitary axis is termed secondary hypogonadism. Dual or mixed forms can also occur. If an insufficient amount of testosterone reaches the target tissues, manifestations of testosterone deficiency may appear in the liver, muscle, and adipose tissue. Testosterone binds directly to the androgen receptor. In the liver, testosterone enhances protein synthesis, while in muscle, testosterone enhances muscle mass. Given its role in muscle, men with reduced testosterone levels may complain of muscle weakness, lethargy, or decreased energy.
In other tissues, testosterone must first undergo conversion before becoming biologically active. Testosterone is converted by aromatization to estradiol in the brain and bone, which then binds to the estrogen receptor. Testosterone enhances bone development by promoting bone accretion. Men with reduced testosterone levels may develop osteoporosis. Testosterone acts in the brain to stabilize mood, enhance libido and may even positively affect cognition. As a result, men with testosterone deficiency can experience mood changes, a lack of motivation, and reduced libido. To bind to the androgen receptor on the skin, hair, gonadal, and prostate tissues, testosterone is converted by 5-ᾳ-reductase to dihydrotestosterone or DHT. Testosterone supports the growth of facial, body, axillary and pubic hair in the adult.
However, in some genetically susceptible men, testosterone may also inhibit hair growth in some areas of the scalp, leading to baldness. In the sexual organs, testosterone contributes to penile growth, spermatogenesis, and prostate growth and function. Profound hypogonadism interferes with the vascular function of the penis, or corpora, and may lead to erectile dysfunction and a decrease in orgasm quality. Other effects of testosterone include the promotion of erythropoiesis and the regulation of immune function. In summary, testosterone production plays a key role in many body tissues, and testosterone deficiency can result in myriad clinical manifestations.
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Nelson Vergel is a chemical engineer who started his search for cutting-edge health knowledge when he was diagnosed with HIV over 30 years ago. Instead of giving into the hopelessness of the deadly diagnosis, he pushed himself to explore therapies to combat wasting syndrome to save his life and those of his peers. He reviewed medical literature and attended numerous health conferences, eventually becoming an advocate member in the National Health Institute (NIH), pharmaceutical research committees, and FDA review panels. To spread survival knowledge, he has given health-related lectures, providing over 700 of them since 1987. Nelson has created popular health forums with over 55,000 members, a health podcast , numerous videos, and has networked with many progressive clinicians. He has been a speaker for 14 pharmaceutical companies on hormones...
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